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RP1 rabbit pAb
RP1 rabbit pAb
- 中文名称:
- RP1 rabbit pAb
- 英文名称:
- RP1 rabbit pAb
- 品牌:
- AAA Biotech
- 品牌介绍:
- AAA Biotech专注于为全球生命科学研究提供高品质的蛋白质研究工具,核心产品包括经严格验证的抗体、重组蛋白及ELISA试剂盒。
- 货号:
- AAA269138
- 规格:
- 0.1 mL|0.1 mL (AF750)|0.1 mL (AP)|0.1 mL (AF635)|0.1 mL (AF610)
- 保存建议:
- Store at -20°C for 1 year
- 货期:
- 6-8周
- 来源宿主:
- Rabbit
- 反应种属:
- Human, Mouse
- 应用:
- IP (Immunoprecipitation), IHC (Immunohistochemistry), ELISA, WB (Western Blot)
- 免责声明:
- *本产品仅供科研实验使用,不得用于临床诊断。*
- 其他:
克隆性:Polyclonal
同型:IgG
克隆号:N/A
特异性:This antibody detects endogeneous level of RP1 at Human/Mouse.
纯度:The antibody was affinity-purified from rabbit serum by affinity-chromatography using specific immunogen.
形式:Liquid in PBS containing 50% glycerol, 0.5% BSA and 0.02% sodium azide. New type preservative N.
浓度:1 mg/mL
- 说明书:
Background: This gene encodes a member of the doublecortin family. The protein encoded by this gene contains two doublecortin domains, which bind microtubules and regulate microtubule polymerization. The encoded protein is a photoreceptor microtubule-associated protein and is required for correct stacking of outer segment disc. This protein and the RP1L1 protein, another retinal-specific protein, play essential and synergistic roles in affecting photosensitivity and outer segment morphogenesis of rod photoreceptors. Because of its response to in vivo retinal oxygen levels, this protein was initially named ORP1 (oxygen-regulated protein-1). This protein was subsequently designated RP1 (retinitis pigmentosa 1) when it was found that mutations in this gene cause autosomal dominant retinitis pigmentosa. Mutations in this gene also cause autosomal recessive retinitis pigmentosa. Transcript variants resulted from an alternative promoter and alternative splicings have been found, which overlap the current reference sequence and has several exons upstream and downstream of the current reference sequence. However, the biological validity and full-length nature of some variants cannot be determined at this time.[provided by RefSeq, Sep 2010].